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Topics:
Allergy & Immunology
•
Urticaria/Angioedema
How often do you rely on using the C1 inhibitor functional assay versus quantitative level alone to diagnosis HAE?
Related Questions
How do you reassure families that no allergy testing is needed for urticaria?
What medications do you counsel patients to avoid if they develop ACEi angioedema?
How do you manage recurrent urticarial lesions in a patient with underlying connective tissue disease that is otherwise well-controlled?
What treatments do you consider for cholinergic urticaria refractory to high dose H1 blockers and omalizumab?
Do you start omalizumab in childbearing patients with CSU?
Is there a role for phenotyping patients with CSU outside of a research setting by ordering IgG-anti-TPO, IgG-anti-FceRI , total IgE, total IgA etc.?
How do you interpret high C1q binding assay with otherwise normal C1q, C3, C4, CH50 in a patient with recurrent urticaria with positive ANA at high titer 1:1280, negative dsDNA, RNP, SM, normal CBC, CMP, UA, and UPCR.
How do you approach work up for underlying rheumatologic disease in patients referred for chronic urticaria?
What is your experience managing patients with chronic spontaneous urticaria occurring only at night?
How do you approach performing a food challenge in a patient with CSU who is dependent on their H1 blockers?
