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Please select the option that best describes you:
Topics:
Internal Medicine
•
Allergy & Immunology
•
Urticaria/Angioedema
How often do you rely on using the C1 inhibitor functional assay versus quantitative level alone to diagnosis HAE?
Related Questions
How do you reassure families that no allergy testing is needed for urticaria?
Do you start omalizumab in childbearing patients with CSU?
What medications do you counsel patients to avoid if they develop ACEi angioedema?
What is your experience managing patients with chronic spontaneous urticaria occurring only at night?
Is there a role for phenotyping patients with CSU outside of a research setting by ordering IgG-anti-TPO, IgG-anti-FceRI , total IgE, total IgA etc.?
How do you approach performing a food challenge in a patient with CSU who is dependent on their H1 blockers?
Is there a role for genetic testing after diagnosing HAE?
What treatments do you consider for cholinergic urticaria refractory to high dose H1 blockers and omalizumab?
What is your preferred prophylactic agent in idiopathic angioedema?
How do you interpret high C1q binding assay with otherwise normal C1q, C3, C4, CH50 in a patient with recurrent urticaria with positive ANA at high titer 1:1280, negative dsDNA, RNP, SM, normal CBC, CMP, UA, and UPCR.
