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Please select the option that best describes you:
Topics:
Rheumatology
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Rheumatoid Arthritis
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TNF Inhibitors
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ILD
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CTD-ILD
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Pulmonary
Do you avoid the use of TNF inhibitors in patients with RA-ILD?
If patient has mild ILD, but active joint disease could this be considered?
Related Questions
How would you approach treatment of active inflammatory arthritis in a patient with MCTD (+RNP/+Chromatin) and stable ILD who has failed MMF, azathioprine and methotrexate?
Would you add abatacept to treat active inflammatory arthritis in a patient with history of RA-ILD who is already taking mycophenolate?
In a patient with low titer +anti-SAE antibody and known ILD, but no other clinical features of dermatomyositis, how would you approach further testing or would you treat the patient as dermatomyositis associated ILD?
Would you initiate antifibrotic therapy in a patient with CTD-ILD experiencing worsening symptoms and declining lung function, despite no clear evidence of fibrosis on CT scans?
How do you approach treatment selection in patients with rheumatoid arthritis and severe COPD?
What is your treatment approach for a young female who has an idiopathic small right elbow effusion with synovial thickening that has resulted in limitation in her range of motion?
What is the maximum dose of leucovorin that you use with methotrexate?
How do you approach immunosuppression in patients with a positive Interferon Gamma Release Assay and prior intravesicular BCG treatment for bladder cancer?
What is your approach to differentiating RA-ILD from medication toxicity (I.e. from methotrexate)?
When considering a biologic for seropositive RA after failure of methotrexate or triple therapy, do you consider using abatacept as a first line biologic or would prefer choosing TNFi?