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Topics:
Cardiology
•
Cardiac Electrophysiology
How does outflow tract VT differ in management between structurally normal and structurally abnormal hearts ?
Related Questions
Do you use DOAC in patients with mild or moderate rheumatic mitral stenosis?
What is the most updated consensus regarding the use of pill in the pocket oral anticoagulation in paroxysmal atrial fibrillation, and populations of patients who are most likely to be considered for enrollment in clinical trials?
How do you advise patients with paroxysmal atrial fibrillation regarding their caffeine consumption, given that a randomized clinical trial found no association between caffeine intake and the triggering of AF episodes?
What would be your threshold to recommend TEE guided DCCV in a patient who has remained in atrial fibrillation in the post-operative period following CABG, who has achieved adequate amiodarone loading dose?
If a patient has potential arrhythmic-sounding syncope and a noninducible type 2 or 3 Brugada ECG pattern, have we excluded Brugada syndrome as the etiology for their syncope?
In which situations would you consider the LOT-CRT approach over CRT or conduction system pacing alone?
Should presence of coronary artery calcifications on CT/CTA be considered as presence of vascular disease on CHA2DS2-VASc score?
Do you typically include exercise restrictions and/or alcohol intake restrictions in routine counseling for patients with atrial fibrillation?
When would you consider AV nodal ablation in CRT-non-responders with persistent atrial fibrillation?
Is there is enough data to recommend LOT-CRT upgrade in CRT nonresponders with a residual wide QRS width assuming the patient had a good LV endocardial-CS lead placement ?
