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Topics:
Internal Medicine
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Rheumatology
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Crystal Arthritis
When would you recommend uric acid-lowering therapy for a patient with asymptomatic hyperuricemia without comorbidities but with family history of gout?
Related Questions
Do you prefer allopurinol or febuxostat for patients with chronic kidney disease who are receiving treatment for asymptomatic hyperuricemia?
If a patient who has tolerated allopurinol for a prolonged period of time is subsequently found to be positive for the HLA-B*58:01 gene, how would you manage urate-lowering therapy thereafter?
Do you prefer using losartan in your patients with hypertension and gout due to its uricosuric effects?
Is there any expanded diagnostic workup that you pursue for a young female patient with gout?
Are there adverse consequences of suppressing serum urate levels too much?
Why are gout flares common in hospitalized patients undergoing diuresis but not commonly observed in cancer patients with hyperuricemia resulting from tumor lysis syndrome?
Can Milwaukee shoulder present with a large subacromial bursitis, or does it predominantly cause joint effusion/destruction?
How do you approach using DMARDs for patients with CPP arthritis who have frequent flares?
Do you routinely recommend barbotage for calcific tendinopathy of the shoulder?
Do you avoid hyaluronic acid injections in patients with chondrocalcinosis on imaging?
