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Topics:
Rheumatology
•
Crystal Arthritis
When would you recommend uric acid-lowering therapy for a patient with asymptomatic hyperuricemia without comorbidities but with family history of gout?
Related Questions
Do you prefer allopurinol or febuxostat for patients with chronic kidney disease who are receiving treatment for asymptomatic hyperuricemia?
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How do you approach using DMARDs for patients with CPP arthritis who have frequent flares?
Is there any expanded diagnostic workup that you pursue for a young female patient with gout?
Do you delay pegloticase in a patient with an active gout flare?
If a patient who has tolerated allopurinol for a prolonged period of time is subsequently found to be positive for the HLA-B*58:01 gene, how would you manage urate-lowering therapy thereafter?
Why are gout flares common in hospitalized patients undergoing diuresis but not commonly observed in cancer patients with hyperuricemia resulting from tumor lysis syndrome?
How would you manage gout with hyperuricemia >10 mg/dl despite the maximum dose of allopurinol plus probenecid and a prior allergic reaction to pegloticase?
Are there adverse consequences of suppressing serum urate levels too much?
